Contact:
University of Arizona College of Medicine - Phoenix 475 North 5th Street, Phoenix, AZ 85004
Associate Professor, N/A
Associate Professor, Tenure - Basic Medical Sciences
Associate Professor, N/A - Psychiatry
Associate Professor, N/A - Translational Neurosciences
Faculty
Basic Medical Sciences; Psychiatry; Translational Neurosciences
Education
- Postdoctoral Fellowship: Neuroscience, Mount Sinai School of Medicine, 2014
- PhD: Neuroscience, Stanford University, 2008
Publications
- Kim HD, Call T, Magazu S & Ferguson D (2017). Drug Addiction and Histone Code Alterations. Adv Exp Med Biol. Vol. 978, 127-143., 01/2017 - Article, Refereed Journal -
- Ferguson D (2017). Cocaine Mediates the Cellular Mechanism of Satiation. Biol Psychiatry. Vol. 81(7), e47-e48., 04/01/2017 - Article, Refereed Journal -
- Kim HD, Hesterman J, Call T, Magazu S, Keeley E, Armenta K, Kronman H, Neve RL, Nestler EJ & Ferguson D (2016). SIRT1 mediates depression-like behaviors in the nucleus accumbens. J Neurosci. Vol. 36(32), 8441-8452., 08/10/2016 - Article, Refereed Journal -
- Ferguson D, Shao N, Heller E, Feng J, Neve R, Kim HD, Call T, Magazu S, Shen L & Nestler EJ (2015). SIRT1-FOXO3a regulate cocaine actions in the nucleus accumbens. J Neurosci. Vol. 35(7), 3100-11., 02/18/2015 - Journal Article -
- Feng J, Wilkinson M, Liu X, Purushothaman I, Ferguson D, Vialou V, Maze I, Shao N, Kennedy P, Koo J, Dias C, Laitman B, Stockman V, LaPlant Q, Cahill ME, Nestler EJ & Shen L (2015). Erratum to: Chronic cocaine-regulated epigenomic changes in mouse nucleus accumbens. Genome Biol. Vol. 16, 227., 10/14/2015 - Article, Refereed Journal -
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Research Interests
- Transcriptomics - Neurodegenerative Diseases - Gene environment interactions and epigenetics - Developmental, cell and molecular biology - Depression
Research Summary
Dr. Ferguson's research program integrates a wide range of molecular and behavioral approaches. Currently, we are evaluating the role of SIRT1 and its downstream targets as potential new candidates for the treatment of neuropsychiatric disorders by performing chromatin immunoprecipitation followed by genome-wide profiling (ChIP-seq) in nucleus accumbens (NAc) tissue from control and socially defeated stressed mice.