- Post-Doctoral: Neuroscience – Epilepsy, Parkinson's Disease, Stanford and Calgary University, 2002 – 2008.
- PhD: Queen's University, 2001.
- Anderson TR, Huguenard JR, Prince DA (in preparation). Site specific regulation of synaptic transmission in fast-spiking cortical interneurons by Na+/K+ ATPase.
- Anderson TR, Huguenard JR, Prince DA (in preparation). The differential effect of Na+/K+ ATPase blockade on cortical layer V neurons.
- Kiss ZHT, Anderson TR. (2007) Cellular mechanisms of action of therapeutic brain stimulation, in Bronzino J and DiLorenzo DJ (Eds): Neuroengineering, CRC Press/Taylor and Francis.
- Iremonger KJ, Anderson TR, Hu B, Kiss ZH. (2006) Cellular mechanisms preventing sustained activation of cortex during subcortical high-frequency stimulation. J Neurophysiol. 96(2): 613-621.
- Anderson TR, Hu B, Iremonger K, Kiss ZH (2006). Selective attenuation of afferent synaptic transmission as a mechanism of thalamic deep brain stimulation-induced tremor arrest. J Neurosci. 26(3):841-850.
For a complete listing of Dr. Anderson's publications, search PubMed.
Migraine, traumatic brain injury, neurophysiology
Dr. Anderson’s laboratory is focused on understanding how mechanisms that regulate cortical excitability influence the development of neurological disease. Specifically, my laboratory utilizes electrophysiological, neuroimaging, optogenetic and behavioral approaches to examine the interface between traumatic brain injury (TBI), migraine and epilepsy. While with more severe TBI the network is predisposed to the development of epilepsy, many patients that experience more mild TBI develop long-term persistent headaches, especially migraine. The underlying mechanisms of post-traumatic epilepsy (PTE) and post-traumatic headache (PTH) are poorly understood and difficult to treat. My laboratory aims to improve understanding of these disease and identify and develop new efficacious therapies.