Shenfeng Qiu

Contact:

University of Arizona College of Medicine - Phoenix ABC-1 Building 425 North 5th Street, Phoenix, AZ 85004
Building
AZ Biomedical Collaborative 1
Associate Professor
Associate Professor - Basic Medical Sciences
Faculty
Basic Medical Sciences

Education

  • Postdoctoral Fellowship: Neurodevelopmental Disorders, Vanderbilt University, 2012
  • PhD: Environmental Toxicology and Neuroscience, University of California, Riverside, 2004
  • MD: Nanjing Medical University, 1994
  • MPH: Nanjing Medical University, 1997

Representative Recent Publications

  • Li H, Zhou X, Li Y, Ma X, Gonzales RJ, Qiu S, Shi FD & Liu Q (2019). The selective sphingosine 1-phosphate receptor 1 modulator RP101075 improves microvascular circulation after cerebrovascular thrombosis. FASEB J. Vol. 33(10), 10935-10941.
  • Ma Z, Gao F, Larsen B, Gao M, Luo Z, Chen D, Ma X, Qiu S, Zhou Y, Xie J, Xi ZX & Wu J (2019). Mechanisms of cannabinoid CB 2 receptor-mediated reduction of dopamine neuronal excitability in mouse ventral tegmental area. EBioMedicine. Vol. 42, 225-237.
  • Huang G, Chen S, Chen X, Zheng J, Xu Z, Doostparast Torshizi A, Gong S, Chen Q, Ma X, Yu J, Zhou L, Qiu S, Wang K & Shi L (2019). Uncovering the Functional Link Between SHANK3 Deletions and Deficiency in Neurodevelopment Using iPSC-Derived Human Neurons. Front Neuroanat. Vol. 13, 23.
  • Stephany CÉ, Ma X, Dorton HM, Wu JC, Solomon AM, Frantz MG, Qiu S & McGee AW (2018). Distinct Circuits for Recovery of Eye Dominance and Acuity in Murine Amblyopia. Curr Biol. Vol. 28(12), 1914-1923.
  • Yang X, Ren H, Wood K, Li M, Qiu S, Shi FD, Ma C & Liu Q (2018). Depletion of microglia augments the dopaminergic neurotoxicity of MPTP. FASEB J. Vol. 32(6), 3336-3345.
Research Interests
Neurodevelopmental Disorders, Electrophysiology, Brain Circuits
Research Summary
The overall interest of Dr. Shenfeng Qiu's lab is to understand the brain origins of neurodevelopmental and neuropsychiatric disorders, such as autism spectrum disorders. One ongoing project (R01MH111619) focuses on the role of MET tyrosine kinase, identified as a major risk for autism. Our lab aims to identify mechanisms by which MET affects neuronal growth, maturation and brain circuit function. We are also interested in the UBE3A protein in Angelman syndrome.